Search results for " Caspase 9"

showing 3 items of 3 documents

A possible biomarker for methadone related deaths

2017

Abstract Methadone (MTH) concentrations in those dying of MTH toxicity totally overlap concentrations where the presence of MTH is only an incidental finding, making it very difficult to make distinctions in actual cases. A biomarker, be it anatomical or biochemical for MTH toxicity is badly needed, particularly if that markers were known to disrupt effective ventilation. Because the brainstem houses the regulatory centers for cardiorespiratory-control enters, it would seem to be the most likely anatomical site to seek abnormalities in cardiorespiratory control. Objective To locate and describe the cells of nucleus of the solitary tract (TS)(NTS) in human brainstem and determine if neuronal…

MalePathologyNecrosisApoptosisAutopsyCohort Studies0302 clinical medicineRetrospective StudieMedicineForensic PathologyNeuronsPoisoningSolitary tractGeneral MedicineRostral ventrolateral medullaNecrosiImmunohistochemistryCaspase 9Narcotic030220 oncology & carcinogenesisToxicityFemaleBrainstemmedicine.symptomBrainstemCaspase-9HumanNarcoticsAdultProgrammed cell deathmedicine.medical_specialty2734Pathology and Forensic MedicineNecrosisForensic ToxicologyYoung Adult03 medical and health sciencesSettore MED/43 - Medicina LegaleSolitary NucleuSolitary NucleusNeurotoxicityHumansRetrospective Studiesbusiness.industryfungiApoptosiBiomarkerNeuronApoptosisApoptosis; Biomarker; Brainstem; Caspase-9; Methadone; Neurotoxicity; Adult; Apoptosis; Brain Stem; Caspase 9; Cohort Studies; Female; Forensic Pathology; Forensic Toxicology; Humans; Immunohistochemistry; Male; Methadone; Narcotics; Necrosis; Neurons; Poisoning; Retrospective Studies; Solitary Nucleus; Young Adult; 2734; LawCohort StudiebusinessLawMethadone030217 neurology & neurosurgeryBrain Stem
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GD3 ganglioside directly targets mitochondria in a bcl-2-controlled fashion.

2000

Lipid and glycolipid diffusible mediators are involved in the intracellular progression and amplification of apoptotic signals. GD3 ganglioside is rapidly synthesized from accumulated ceramide after the clustering of death-inducing receptors and triggers apoptosis. Here we show that GD3 induces dissipation of DeltaPsim and swelling of isolated mitochondria, which results in the mitochondrial release of cytochrome c, apoptosis inducing factor, and caspase 9. Soluble factors released from GD3-treated mitochondria are sufficient to trigger DNA fragmentation in isolated nuclei. All these effects can be blocked by cyclosporin A, suggesting that GD3 is acting at the level of the permeability tran…

Programmed cell deathCeramideApoptosisMitochondria LiverMitochondrionliverBiochemistryMembrane Potentialschemistry.chemical_compoundGangliosidesGeneticsAnimalsMolecular BiologySettore MED/04 - Patologia GeneralebiologyCytochrome cCaspase 9SialyltransferasesCell biologyRatsmitochondriaEnzyme ActivationchemistryMitochondrial permeability transition poreProto-Oncogene Proteins c-bcl-2ApoptosisCaspasesbiology.proteinCyclosporinecaspases; cyclosporine; proto-oncogene proteins c-bcl-2; sialyltransferases; caspase 9; rats; animals; enzyme activation; apoptosis; membrane potentials; gangliosides; mitochondria liver; subcellular fractionsApoptosis-inducing factorlipids (amino acids peptides and proteins)ApoptosomeBiotechnologySubcellular FractionsFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Orthodontic Stress BCL-2 Nodulation and Human Odontoblast's Survival

2013

Settore BIO/17 - IstologiaBcl-2 Caspase 9 Odontoblasts
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